Apoptosis (programmed cell death) is required for the removal of infected, damaged or unwanted cells. Inadequate cell death is a major contributing factor to tumorigenesis, while excess cell death contributes to neurodegeneration and autoimmune diseases. Current investigations suggest the mechanisms by which Bcl-2 might pre-vent cell death. Bcl-2 family members mediate anti-apoptotic signals in a wide variety of human cell systems. Bcl-2 protein family, through its role in the regu-lation of apoptotic pathways, is possibly related to cancer pathophysiology. This review article discribes some pathways how apoptotic cell death is controlled by this protein family
AbstractThe Bcl-2 family consists of about 20 homologues of important pro- and anti-apoptotic regula...
BCL-2 was the first anti-death gene discovered, a milestone with far reaching implications for tumor...
The ability of a cell to undergo mitochondrial apoptosis is governed by pro- and anti-apoptotic memb...
Programmed cell death, apoptosis, plays an integral role in a variety of biological events, e.g. mor...
The Bcl-2 protein family consists of about 20 homologues important for stimulation and inhibition of...
THESIS 8947Apoptosis is a highly controlled mechanism of cell suicide that is activated in response ...
Apoptosis (or programmed cell death) is a genetically controlled “cell suicide” pathway which plays ...
AbstractBcl-2 family proteins are key intracellular regulators of programmed cell death. Several rec...
The literature on apoptosis has grown tremendously in recent years, and the mechanisms that are invo...
The literature on apoptosis has grown tremendously in recent years, and the mechanisms that are invo...
Apoptosis, programmed cell death, has a central role in developmental biology and in maintaining the...
Bcl-2 is a protein that inhibits apoptosis, leading to cell survival. The Bcl-2 family has six diffe...
(A) The BCL-2-regulated apoptotic pathway involves the following proteins pro-survival BCL-2 protein...
The disturbance of apoptosis molecular signaling pathways is involved in carcinogenesis. BCL2 family...
The Bcl-2 protein blocks a distal step in an evolutionarily conserved pathway for programmed cell de...
AbstractThe Bcl-2 family consists of about 20 homologues of important pro- and anti-apoptotic regula...
BCL-2 was the first anti-death gene discovered, a milestone with far reaching implications for tumor...
The ability of a cell to undergo mitochondrial apoptosis is governed by pro- and anti-apoptotic memb...
Programmed cell death, apoptosis, plays an integral role in a variety of biological events, e.g. mor...
The Bcl-2 protein family consists of about 20 homologues important for stimulation and inhibition of...
THESIS 8947Apoptosis is a highly controlled mechanism of cell suicide that is activated in response ...
Apoptosis (or programmed cell death) is a genetically controlled “cell suicide” pathway which plays ...
AbstractBcl-2 family proteins are key intracellular regulators of programmed cell death. Several rec...
The literature on apoptosis has grown tremendously in recent years, and the mechanisms that are invo...
The literature on apoptosis has grown tremendously in recent years, and the mechanisms that are invo...
Apoptosis, programmed cell death, has a central role in developmental biology and in maintaining the...
Bcl-2 is a protein that inhibits apoptosis, leading to cell survival. The Bcl-2 family has six diffe...
(A) The BCL-2-regulated apoptotic pathway involves the following proteins pro-survival BCL-2 protein...
The disturbance of apoptosis molecular signaling pathways is involved in carcinogenesis. BCL2 family...
The Bcl-2 protein blocks a distal step in an evolutionarily conserved pathway for programmed cell de...
AbstractThe Bcl-2 family consists of about 20 homologues of important pro- and anti-apoptotic regula...
BCL-2 was the first anti-death gene discovered, a milestone with far reaching implications for tumor...
The ability of a cell to undergo mitochondrial apoptosis is governed by pro- and anti-apoptotic memb...
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