Add to ORKG), a potent vasodilator, has its greatest ef-fects on the uterine vasculature, blood flow (UBF) increas-ing 10-fold. The mechanism(s) responsible for E 2 b-induced vasodilation is unclear. We determined if nitric oxide (NO)-induced increases in cGMP modulate estrogen-induced in-creases in UBF, and if cyclooxygenase inhibition modifies E 2 b responses. Nonpregnant ( n
Few studies have examined the potential effects of childbirth on the responses of the female vascula...
We have previously shown that estrogen exerts a vasoprotective effect by accelerating reendotheliali...
1. The L-arginine-NO pathway has been implicated in the vasorelaxant effect of 17-beta-oestradiol. H...
Nitric oxide synthase (NOS) contributes to estradiol-17ß (E2ß)-induced uterine vasodilation, but add...
Normal pregnancy is associated with dramatic increases in uterine blood flow to facilitate the bidir...
Covalent adduction of a NO moiety to cysteines (S-nitrosylation or SNO) is a major route for NO to d...
Regulation of arterial tone relies heavily on molecular crosstalk between endothelial and smooth mus...
The endogenous estradiol metabolite 2-methoxyestradiol (2-ME) reduces atherosclerotic lesion formati...
G-protein-coupled estrogen receptors (GPERs) have been proposed to mediate estrogen-mediated vasodil...
The regulation of vascular tone in the uterine circulation is a key determinant of appropriate utero...
INTRODUCTION 17β-estradiol (E2) has been found to induce vasodilation in the cardiovascular syste...
Numerous studies have identified estrogens as a driving force that regulates uterine vasodilation du...
Nitric oxide (NO) is known to be an important relaxant of contractile activity in various muscles in...
Cardiovascular disease is the leading cause of morbidity and mortality in postmenopausal women. EM-6...
The effects of estrogen on the female reproductive system are well known. In contrast, comparatively...
Few studies have examined the potential effects of childbirth on the responses of the female vascula...
We have previously shown that estrogen exerts a vasoprotective effect by accelerating reendotheliali...
1. The L-arginine-NO pathway has been implicated in the vasorelaxant effect of 17-beta-oestradiol. H...
Nitric oxide synthase (NOS) contributes to estradiol-17ß (E2ß)-induced uterine vasodilation, but add...
Normal pregnancy is associated with dramatic increases in uterine blood flow to facilitate the bidir...
Covalent adduction of a NO moiety to cysteines (S-nitrosylation or SNO) is a major route for NO to d...
Regulation of arterial tone relies heavily on molecular crosstalk between endothelial and smooth mus...
The endogenous estradiol metabolite 2-methoxyestradiol (2-ME) reduces atherosclerotic lesion formati...
G-protein-coupled estrogen receptors (GPERs) have been proposed to mediate estrogen-mediated vasodil...
The regulation of vascular tone in the uterine circulation is a key determinant of appropriate utero...
INTRODUCTION 17β-estradiol (E2) has been found to induce vasodilation in the cardiovascular syste...
Numerous studies have identified estrogens as a driving force that regulates uterine vasodilation du...
Nitric oxide (NO) is known to be an important relaxant of contractile activity in various muscles in...
Cardiovascular disease is the leading cause of morbidity and mortality in postmenopausal women. EM-6...
The effects of estrogen on the female reproductive system are well known. In contrast, comparatively...
Few studies have examined the potential effects of childbirth on the responses of the female vascula...
We have previously shown that estrogen exerts a vasoprotective effect by accelerating reendotheliali...
1. The L-arginine-NO pathway has been implicated in the vasorelaxant effect of 17-beta-oestradiol. H...