RESEARCH Energy replacement using

Full list of author information is available at the end of the articlewomen [1-3]. Moreover, increased postprandial TG con-centrations also appear to promote blood remnant choles-terol [4], thereby indirectly contributing to atherosclerosis [5]. It has been shown that a single bout of aerobic exercise can decrease postprandial TG concentrations [6-8]. The energy expenditure of exercise is postulated to be a more important determinant behind reductions in postprandial accumulated in short bouts throughout the day or a single longer bout as long as the energy expenditure of exercise is sufficient [8]. Whilst the energy expenditure of aerobic exercise has been demonstrated to be related to the reduction in postprandial lipaemia [7] the relationship between en-ergy deficits and reductions in postprandial TG concen-trations is not clearly defined [11]. An exercise-induced energy deficit results in a greater reduction in postpran-dial TG concentration than a dietary-induced energy deficit [12]. Conversely, post-exercise restoration of an * Correspondence