[1]. The bleeding is caused by either inactivation or ex- defect size, tension, and mobility that affect the rates of

is available at the end of the articlecess consumption of platelets and/or clotting factors be-cause of increased fibrinolysis and may have a gradual or acute onset. Once this process starts, changes in the pa-tient’s status occur rapidly and require intense monitor-ing with subsequent changes in therapy as indicated [2]. The classic cutaneous manifestations of DIC present as skin wounds such as bruising, contusions, petechiae, purpura, acral cyanosis, hemorrhagic bullae, purpura healing and residual scarring [4]. Platelet rich plasma (PRP) not only enhances wound healing but also helps regenerate skin tissue [5]. Autologous PRP enhances wound healing and tissue regeneration and has been used as a source of growth factors in bone fracture treatment and in general wound healing for skin defects or dental mucosal wound care [6-8]. However, the current method of applying PRP for skin recovery used in veterinary stud-ies is limited to autologous PRP applications [9,10]. This report describes a very small patient (toy breed, < 2 kg) with a large skin defect including the tail and subsequent use of topical allogeneic PRP as adjunct * Correspondence