Add to ORKGCopyright © 2015 Marc Schaber et al. This is an open access article distributed under the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited. Introduction. The aim of the present study was to investigate whether a 12-hour exposure in a normobaric hypoxic chamber would induce changes in the hemostatic system and a procoagulant state in volunteers suffering from acute mountain sickness (AMS) and healthy controls. Materials and Methods. 37 healthy participants were passively exposed to 12.6 % FiO 2 (simulated altitud
International audiencePerforming exercise during the first hours of hypoxic exposure is thought to e...
This study examined whether hypoxia causes free radical-mediated disruption of the blood-brain barri...
Background: This study aimed to observe the effects of a fast acute ascent to simulated high altitud...
Introduction. The aim of the present study was to investigate whether a 12-hour exposure in a normob...
The present study was undertaken to determine the integrative physiology behind acute mountain sickn...
We examined the control of breathing, cardio-respiratory effects and the prevalence of acute mountai...
Hypoxia (oxygen deprivation) is known to be associated with deep vein thrombosis and venous thromboe...
Objective Normobaric (NH) and hypobaric hypoxia (HH) are associated with acute mountain sickness (A...
The purpose of this study was to determine whether there is a difference in hemodynamic responses to...
Hypoxia (oxygen deprivation) is known to be associated with deep vein thrombosis and venous thromboe...
We tested the hypothesis that dynamic cerebral autoregulation (CA) and blood–brain barrier (BBB) fu...
Acute mountain sickness (AMS) is caused by rapid ascent to altitude (>2500 m) and remains a poorly u...
Hypoxia (oxygen deprivation) is known to be associated with deep vein thrombosis and venous thromboe...
International audiencePerforming exercise during the first hours of hypoxic exposure is thought to e...
This study examined whether hypoxia causes free radical-mediated disruption of the blood-brain barri...
Background: This study aimed to observe the effects of a fast acute ascent to simulated high altitud...
Introduction. The aim of the present study was to investigate whether a 12-hour exposure in a normob...
The present study was undertaken to determine the integrative physiology behind acute mountain sickn...
We examined the control of breathing, cardio-respiratory effects and the prevalence of acute mountai...
Hypoxia (oxygen deprivation) is known to be associated with deep vein thrombosis and venous thromboe...
Objective Normobaric (NH) and hypobaric hypoxia (HH) are associated with acute mountain sickness (A...
The purpose of this study was to determine whether there is a difference in hemodynamic responses to...
Hypoxia (oxygen deprivation) is known to be associated with deep vein thrombosis and venous thromboe...
We tested the hypothesis that dynamic cerebral autoregulation (CA) and blood–brain barrier (BBB) fu...
Acute mountain sickness (AMS) is caused by rapid ascent to altitude (>2500 m) and remains a poorly u...
Hypoxia (oxygen deprivation) is known to be associated with deep vein thrombosis and venous thromboe...
International audiencePerforming exercise during the first hours of hypoxic exposure is thought to e...
This study examined whether hypoxia causes free radical-mediated disruption of the blood-brain barri...
Background: This study aimed to observe the effects of a fast acute ascent to simulated high altitud...