doi: 10.3389/fncel.2014.00269 C1-Inhibitor protects from focal brain trauma in a cortical cryolesion mice model by reducing thrombo-inflammatio
BACKGROUND AND PURPOSE: The restoration of blood-flow following cerebral ischemia incites a series o...
Ischemia cerebral stroke is one of the common neurological diseases with severe inflammatory respons...
Although traumatic brain injury (TBI) acutely disrupts the cortex, most TBI-related disabilities ref...
Traumatic brain injury (TBI) induces a strong inflammatory response which includes blood-brain barri...
Inflammation and thrombosis are pathophysiological hallmarks of ischemic stroke still unamenable to ...
BACKGROUND: The goal of the study was to evaluate the effects of Cl-inhibitor (C1-INH), an endogenou...
OBJECTIVE: The aim of the study was to evaluate the effects of C1-inhibitor (C1-INH), an endogenous ...
Previous studies demonstrated that C1-inhibitor (C1-INH), a complement and contact-kinin systems inh...
C1-inhibitor (C1-INH) is a major regulator of the complement classical pathway. Besides this action,...
BACKGROUND: Although activation of the complement system has been implicated in the progression of h...
Bei einem SHT handelt es sich um eine mechanische Schädigung des Hirngewebes, verursacht durch eine ...
OBJECTIVE: The complement cascade has been implicated in cerebral ischemia/reperfusion injury. To de...
In spite of antibiotic treatment, pneumococcal meningitis continues to be associated with significan...
Background: Neuroinflammation is an important secondary mechanism that is a key mediator of the long...
OBJECTIVE: Ischemic stroke may result from transient or permanent reductions of regional cerebral bl...
BACKGROUND AND PURPOSE: The restoration of blood-flow following cerebral ischemia incites a series o...
Ischemia cerebral stroke is one of the common neurological diseases with severe inflammatory respons...
Although traumatic brain injury (TBI) acutely disrupts the cortex, most TBI-related disabilities ref...
Traumatic brain injury (TBI) induces a strong inflammatory response which includes blood-brain barri...
Inflammation and thrombosis are pathophysiological hallmarks of ischemic stroke still unamenable to ...
BACKGROUND: The goal of the study was to evaluate the effects of Cl-inhibitor (C1-INH), an endogenou...
OBJECTIVE: The aim of the study was to evaluate the effects of C1-inhibitor (C1-INH), an endogenous ...
Previous studies demonstrated that C1-inhibitor (C1-INH), a complement and contact-kinin systems inh...
C1-inhibitor (C1-INH) is a major regulator of the complement classical pathway. Besides this action,...
BACKGROUND: Although activation of the complement system has been implicated in the progression of h...
Bei einem SHT handelt es sich um eine mechanische Schädigung des Hirngewebes, verursacht durch eine ...
OBJECTIVE: The complement cascade has been implicated in cerebral ischemia/reperfusion injury. To de...
In spite of antibiotic treatment, pneumococcal meningitis continues to be associated with significan...
Background: Neuroinflammation is an important secondary mechanism that is a key mediator of the long...
OBJECTIVE: Ischemic stroke may result from transient or permanent reductions of regional cerebral bl...
BACKGROUND AND PURPOSE: The restoration of blood-flow following cerebral ischemia incites a series o...
Ischemia cerebral stroke is one of the common neurological diseases with severe inflammatory respons...
Although traumatic brain injury (TBI) acutely disrupts the cortex, most TBI-related disabilities ref...