Review Article The Beta-Amyloid Protein of Alzheimer’s Disease: Communication Breakdown by Modifying the

Copyright © 2013 Sara H. Mokhtar et al. This is an open access article distributed under the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited. Alzheimer’s disease (AD) is one of the most prevalent severe neurological disorders afflicting our aged population. Cognitive decline, a major symptom exhibited by AD patients, is associated with neuritic dystrophy, a degenerative growth state of neurites. Themolecularmechanisms governing neuritic dystrophy remain unclear.Mounting evidence indicates that theAD-causative agent