Suppression of false recognition in Alzheimer’s disease and in patients with frontal lobe lesions

Previous research has shown that patients with Alzheimer's disease show increasing levels of false rec-ognition across ®ve repeated study±test trials of seman-tic associates. The present study tested the hypotheses that (i) the increasing false recognition was partly due to the frontal lobe dysfunction of patients with Alzheimer's disease, and (ii) a failure of source moni-toring was the central mechanism by which frontal lobe dysfunction led to increasing false recognition across trials. In Experiment 1, patients with frontal lobe lesions and controls were examined in the same repeated trials paradigm as that used previously in patients with Alzheimer's disease. Although controls were able to reduce their false recognition across trials, the patients with frontal lobe lesions were not, an