Abstract: Abscess formation has been viewed as a host defense strategy to contain the spread of infection. However, abscesses are also serious and life-threatening manifestations of persisting micro-bial infection. The initiation of abscess formation, both clinically and experimentally, involves the release of bacteria and an abscess-potentiating agent (e.g., fecal fiber or an analog) into a sterile site, with host defense mechanisms being unable to eliminate the infecting organisms. Abscess forma-tion is aided by a combination of factors that share a common feature: impairment of phagocytic kill-ing and hence clearance of microorganisms. These include bacterial virulence factors (e.g., capsule formation, succinic acid production); compleme...
Objective: To develop a standardised and reproducible model of intra-abdominal infection and abscess...
Initial events and effector mechanisms of most inflammatory and autoimmune diseases remain largely u...
Antimicrobial host defense is dependent on the rapid recruitment of inflammatory cells to the site o...
Intra-abdominal abscesses are a potential source of recurrent or residual infection after surgical i...
Abscess formation associated with secondary peritonitis causes severe morbidity and can be fatal. Fo...
SUMMARY. An examination of 10 strains of Staphylococcus aureus for survival within abscesses develop...
Murine abscesses induced by intraperitoneal injection of a mixture of Escherichia coli, Bacteroides ...
Host defence mechanisms associated with the inhibition of translocation of bacteria from the gastroi...
Summary. A model whereby a known number of coagulase-negative staphylococci were packed into capilla...
Contains fulltext : 19710.pdf (publisher's version ) (Closed access)The role of en...
<div><p>Neutrophil abscess formation is critical in innate immunity against many pathogens. Here, th...
Brain abscess represents a significant medical problem despite recent advances made in detection and...
<p>(A) CCL2 mRNA levels in liver tissue of mice infected with <i>E. histolytica</i> trophozoites (AL...
<p>A: Neck. B: Surgical removal of the abscess site. C: Abdomen. D: Abscess formation in liver and s...
BACKGROUND: Intra-abdominal abscesses are sources of recurrent or ongoing abdominal sepsis. They are...
Objective: To develop a standardised and reproducible model of intra-abdominal infection and abscess...
Initial events and effector mechanisms of most inflammatory and autoimmune diseases remain largely u...
Antimicrobial host defense is dependent on the rapid recruitment of inflammatory cells to the site o...
Intra-abdominal abscesses are a potential source of recurrent or residual infection after surgical i...
Abscess formation associated with secondary peritonitis causes severe morbidity and can be fatal. Fo...
SUMMARY. An examination of 10 strains of Staphylococcus aureus for survival within abscesses develop...
Murine abscesses induced by intraperitoneal injection of a mixture of Escherichia coli, Bacteroides ...
Host defence mechanisms associated with the inhibition of translocation of bacteria from the gastroi...
Summary. A model whereby a known number of coagulase-negative staphylococci were packed into capilla...
Contains fulltext : 19710.pdf (publisher's version ) (Closed access)The role of en...
<div><p>Neutrophil abscess formation is critical in innate immunity against many pathogens. Here, th...
Brain abscess represents a significant medical problem despite recent advances made in detection and...
<p>(A) CCL2 mRNA levels in liver tissue of mice infected with <i>E. histolytica</i> trophozoites (AL...
<p>A: Neck. B: Surgical removal of the abscess site. C: Abdomen. D: Abscess formation in liver and s...
BACKGROUND: Intra-abdominal abscesses are sources of recurrent or ongoing abdominal sepsis. They are...
Objective: To develop a standardised and reproducible model of intra-abdominal infection and abscess...
Initial events and effector mechanisms of most inflammatory and autoimmune diseases remain largely u...
Antimicrobial host defense is dependent on the rapid recruitment of inflammatory cells to the site o...