Add to ORKGThe polyglutamine neurodegenerative protein ataxin-3 binds polyubiquitylated proteins an
Ataxin-3, the disease protein in the neurodegenerative disorder Spinocerebellar Ataxia Type 3 or Mac...
AbstractThe ubiquitin-proteasome system targets numerous cellular proteins for degradation. In addit...
<div><p>Alterations in the ubiquitin-proteasome system (UPS) have been reported in several neurodege...
Spinocerebellar ataxia type 3/Machado Joseph disease is a dominantly inherited neurodegenerative dis...
Spinocerebellar ataxia type 3 is a human neurodegenerative disease resulting from polyglutamine trac...
Deubiquitinating enzymes (DUbs) play important roles in many ubiquitin-dependent pathways, yet how D...
Protein context clearly influences neurotoxicity in polyglutamine diseases, but the contribution of ...
Spinocerebellar ataxia type 3 (SCA3) results from expansion of a glutamine stretch in the disease pr...
Insoluble protein aggregates are consistently found in neurodegenerative disorders caused by expande...
Polyglutamine diseases are a family of neurodegenerative diseases caused by expansion of a CAG repea...
The 26S proteasome is a large (~2.5 MDa) protein complex consisting of at least 33 different subunit...
Ataxin-3 consists of an N-terminal globular Josephin domain and an unstructured C-terminal region co...
Neurodegenerative disorders are a heterogeneous group of clinically and pathologically diverse disea...
:Anomalous expansion of a polyglutamine (polyQ) tract in the protein ataxin-3 causes spinocerebellar...
AbstractAnomalous expansion of a polyglutamine (polyQ) tract in the protein ataxin-3 causes spinocer...
Ataxin-3, the disease protein in the neurodegenerative disorder Spinocerebellar Ataxia Type 3 or Mac...
AbstractThe ubiquitin-proteasome system targets numerous cellular proteins for degradation. In addit...
<div><p>Alterations in the ubiquitin-proteasome system (UPS) have been reported in several neurodege...
Spinocerebellar ataxia type 3/Machado Joseph disease is a dominantly inherited neurodegenerative dis...
Spinocerebellar ataxia type 3 is a human neurodegenerative disease resulting from polyglutamine trac...
Deubiquitinating enzymes (DUbs) play important roles in many ubiquitin-dependent pathways, yet how D...
Protein context clearly influences neurotoxicity in polyglutamine diseases, but the contribution of ...
Spinocerebellar ataxia type 3 (SCA3) results from expansion of a glutamine stretch in the disease pr...
Insoluble protein aggregates are consistently found in neurodegenerative disorders caused by expande...
Polyglutamine diseases are a family of neurodegenerative diseases caused by expansion of a CAG repea...
The 26S proteasome is a large (~2.5 MDa) protein complex consisting of at least 33 different subunit...
Ataxin-3 consists of an N-terminal globular Josephin domain and an unstructured C-terminal region co...
Neurodegenerative disorders are a heterogeneous group of clinically and pathologically diverse disea...
:Anomalous expansion of a polyglutamine (polyQ) tract in the protein ataxin-3 causes spinocerebellar...
AbstractAnomalous expansion of a polyglutamine (polyQ) tract in the protein ataxin-3 causes spinocer...
Ataxin-3, the disease protein in the neurodegenerative disorder Spinocerebellar Ataxia Type 3 or Mac...
AbstractThe ubiquitin-proteasome system targets numerous cellular proteins for degradation. In addit...
<div><p>Alterations in the ubiquitin-proteasome system (UPS) have been reported in several neurodege...